Clinical Report:
Common Clinical and Biological Windows on CFS and Rickettsial Diseases

J Chronic Fatigue Syndrome, Vol. 6, No. 3/4, 2000, pp. 133-145

C.L. Jadin, MD, MBBCh

C.L. Jadin, Postnet Suite 182, Private Bag X3, North Riding, 2162,
Republic of  South Africa (E-mail: mailto:gerinjadin@icon.co.za).

SUMMARY. From 1991, links between CFS and Rickettsial Diseases were
uncovered because of their similar clinical presentation. Further
research linked them even more. Five Rickettsia strains, suspected to be
the cause, or an important factor in CFS, were identified by means of the
Giroud Micro-Agglutination test and were widely found to be positive in
patients’ serum, diagnosed as suffering from CFS, Fibromyalgia,
Rheumatoid Arthritis, Multiple Sclerosis, Depression, Psychosis, Heart
Diseases, and Auto-Immune Diseases. This finding leads us to submit those
originally differently diagnosed patients to the same tetracycline
treatment. This proved to be a great success. The increasing number of
patients gave us the opportunity to establish a biological checklist
(regardless of the diversity of the pathology) of infections, organs’
functions and auto-immune profile. We found the differences in positivity
to depend on four factors: length of illness, virulence of germs,
cohabitation of germs, and the state of the host immune system. These
studies suggest that auto-immune diseases could have an infectious
origin. Better knowledge and mastery of the co-factors would be
determinant in speeding recovery.

KEYWORDS. Intracellular organisms, clinical and biological changes,
cellular anoxemia, CFS

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